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- Bauxite pulmonary fibrosis 铁钒土肺纤维化
- Quercetin could prevent and treat BLM induced pulmonary fibrosis. 槲皮素对博莱霉素致肺纤维化有一定的防治作用
- Pulmonary fibrosis is an important toxic effect of paraquat. 在各种肺组织损伤中又以肺纤维化损伤最为突出。
- The most likely diagnosis is Idiopathic pulmonary fibrosis (IPF). 最可能的诊断是特发性肺纤维化（IPF）。
- Only about 1% of such persons get a significant degree of pulmonary fibrosis. 仅有大约1%25的这种病人出现明显的肺纤维化。
- Objective: Acute idiopathic pulmonary fibrosis (AIPF) is a rare disease. 目的：急性特发性肺间质纤维化是少见疾病。
- The results indicate that rat model of pulmonary fibrosis replicated by BLM is similar to that of mankind. 结果表明：博莱霉素肺纤维化动物模型的肺病理组织学与病理生理学改变与人肺纤维化相似。
- It was similar to the two groups in late reaction to radioactive pulmonary fibrosis. 在减轻放射性肺纤维化等晚反应方面两组相似。
- These morphologic changes provide basis for formation of idiopathic pulmonary fibrosis. 上述形态学改变对特发性肺纤维化的形成提供了一些依据。
- Recent studies on the pathogenesis of paraquat-induced pulmonary fibrosis are reviewed in this paper. 该文就近年来关于百草枯所致肺纤维化发生机制的研究进展进行综述。
- Objective To evaluate the effect of dipyridamole (DPM) and adenosine(ADO) on interstitial pulmonary fibrosis in mice. 目的 通过腺苷 (ADO)受体拮抗剂 茶碱 (TH) ,探讨ADO、双哌达莫 (DPM)对小鼠肺间质纤维化过程的干预作用。
- Objective To observe ultrastructural changes and study the pathogenesis of idiopathic pulmonary fibrosis. 目的观察特发性肺纤维化的超微结构变化并探讨其发病机理。
- Objective To study the role of matrix metalloproteinase-2 (MMP 2) in rat pulmonary fibrosis. 目的 研究基质金属蛋白酶 2 (MMP2 )在肺纤维化中的作用。
- Diagnosis: Idiopathic pulmonary fibrosis, with intralobular interstitial thickening. 诊断：特发性肺纤维化并小叶间质增厚。
- The degree of alveolitis and pulmonary fibrosis in group Flu was improved as compared with that of group BLM. 病理学观察显示 :Flu组肺泡炎和纤维化程度较BLM组均减轻 ,但仍未恢复到正常水平 ;
- Abstract: Objective To observe ultrastructural changes and study the pathogenesis of idiopathic pulmonary fibrosis. 文摘:目的观察特发性肺纤维化的超微结构变化并探讨其发病机理。
- By injecting bleomycin (BLM) into trachea of rats, we successfully established an pulmonary fibrosis animal model. 通过气管内注射博莱霉素(Blcomycin，BLM)的方法复制大鼠肺纤维化动物模型，其普通病理及超微病理改变、动脉血气指标变化及肺组织TGF一p，、FN表达等均符合肺纤维化表现，证明模型复制成功。
- Starting from this bases the question arises, which is the cause of glutathione deficiency in pulmonary fibrosis ? 基于上述情况，我们不禁要问：在肺纤维化疾病中，是什么原因造成谷胱甘肽的不足呢？
- All of this shows that scleroderma patients with pulmonary fibrosis than before treatment significantly improved. 这些都表明硬皮病患者肺间质纤维化的程度较治疗前明显好转。
- Conclusions Mutations in the genes encoding telomerase components can appear as familial idiopathic pulmonary fibrosis. 结论：端粒酶组分的编码基因发生突变时，能以家庭性特发性肺纤维变性的形式出现。
