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- Methods Plasmids containing HPV-16 E6 or E7 were used to generate absolute standard curves. 方法将制备的含HPV-16E6和E7基因的质粒作为标准品,建立实时荧光定量PCR和RT-PCR方法,分别对3种宫颈癌细胞株进行HPV-16E6和E7基因的DNA和RNA拷贝数的定量检测。
- In Large White, the BB genotype at E5 was favorable marker genotype for TNB and NBA, and the BB genotype at E7 was favorable marker genotype for TNB. 对于大白猪,E5位点BB基因型可作为在TNB和NBA性状上的标记基因型,E7位点BB基因型可以作为TNB性状的标记基因型。
- HPV16 E6 and E7 genes may act as risk factors in the development of poor-differentiated esophageal cancer in Kazakh population. 另外,HPV16 E6与E7基因和哈萨克族食管癌病理组织学分级的生物学行为密切相关。
- The malignant phenotype of human papillomavirus (HPV)-positive cancer cells is maintained by the activity of the viral E6 and E7 genes. 基因时代报道: 人乳头状瘤病毒(HPV)阳性癌细胞的恶性表型是通过病毒E6和E7基因的活性得以维持的。
- Follow up 8D / PDCA for BI rejection. 跟进供应商8D/PDCA改善行动。
- Tracking and confirmed 8D and quality issues. 3 8D和质量问题的追踪及确认评估。
- At the end of the year,87 kaitos were in operation. 年底时,共有87艘"街渡"行走。
- Has the team brainstormed the 8D response? 小组成员是否对8D进行过头脑风暴式的集思广益.
- It is supposed to reach 87 degrees Fahrenheit. 可能达到华氏87度。
- The recombinant plasmid pUC18 E6 had been got. 获得重组质粒 pUC18 E6。
- In this examination he got 87 points. 此次考试他得了87分。
- It's supposed to reach 87 degrees Fahrenheit. 可能达到华氏 87 度。
- The conclusion shows: EACC 87%. DBE 6%. EAA 7%. 测定结果表明:EACC含量为87%25;DBE含量为6%25;EAA含量为7%25.
- Disease-free survival(DFS) at 10 years was 87%. 患者10年总生存率为87%25。
- The Saweyers live at 87 King Street. 索耶一家住在王国街87号.
- The Sawyers live in the 87 King Street. 索耶一家住在国王大街87号。
- Methods The expressions of HPV16 E6 and HPV16 E7 oncoproteins were detected by immunohistochemistry in 15 cases of normal cervices,25 cases of CINs and 61 cases of invasive cervical carcinomas. 方法采用免疫组化SP法对15例正常宫颈、25例宫颈上皮内瘤变(CIN)以及61例浸润性宫颈癌组织进行了HPV16 E6、HPV16 E7蛋白表达的检测。
- The Sawyers live in 87 King Street. 索耶一家住在国王街87号。
- Dialog 87 rve got some bad news for you. 我有些坏消息告诉你。
- But recent research has found that: The immune react to E6 and E7 protein of HPV18 have no relation to the regression of pathological changes. This maybe ascribe to the proteins'later expressing in the infected cells. 但最近的研究也发现:机体对高危型HPV病毒(HPV16、HPV18)E6、E7蛋白的免疫反应与病变的自行消退没有必然联系,这或许是因为E6、E7的高表达是在晚期病变组织中,而在感染初期和宫颈上皮内瘤变发生的早期,它们的表达被E2蛋白所抑制。